CANCER AND OBESITY

Obese people often have chronic low-level inflammation which can cause DNA damage that leads to cancer.  Obese patients are more likely than normal-weight individuals to have disorders that cause chronic local inflammation and that are risk factors for the developing of certain type of cancers. Such as Barrett oesophagus, gallbladder cancer .adipose tissue produces excess amounts of estrogen, elevated levels of estrogen leads to increased risks of breast, endometrial, ovarian, and some other cancers. Obese people often have increased insulin levels in blood and insulin-like growth factor-1 (IGF-1). High levels of insulin and IGF-1 promotes the development of colon, kidney, prostate, and endometrial cancers.

Fat cells produce adipokines, hormones that may stimulate or inhibit cell growth. Fat cells have direct and indirect effects on cell growth regulators, which include mammalian target of rapamycin (mTOR) and AMP-activated protein kinase. Other mechanisms by which obesity could affect cancer risk include changes in the mechanical properties of altering immune responses and the scaffolding that surrounds breast cells, which effects on the nuclear factor of kappa beta system and oxidative stress. Obesity may also lead to increased cancer-related mortality.

 

  • Obesity and Endometrium Cancer
  • Obesity and Gall Bladder Cancer
  • Obesity and Pancreatic Cancer
  • Obesity and Prostate Cancer

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